Factors associated with cervical preneoplastic injuries in patients from state-owned medical institution in Aipe, from january to september, 2009
Factores asociados a lesiones preneoplásicas de cuello uterino en usuarias de la ESE del municipio de Aipe, en el periodo enero a septiembre de 2009
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Cervical cancer causes about 500,000 deaths per year worldwide in developing countries; its incidence reaches up to 40 per 100,000 women. An observational, epidemiological, analytical study of cases and controls was carried out in 198 adult women whose age is no more than 55, who attended the cervical cancer early detection program in Aipe, Huila, from January 1 to September 30, 2009. All women with hysterectomy from any cause and those who were minors or who are over 55 years old were excluded from the study. The population was interviewed in relation to gynecological, obstetric and socio-cultural history. The risk of precancerous lesions in the uterus, is increased 5.92 times more in comparison to those women free from infection, having from 6 to 7 sexual partners increases 4.67 times more, compared to those who have had a single sexual partner. Women who have developed vaginal infections have 5.88 times more risk, compared to those who have not suffered from that disease; likewise, having more than five sexual partners increases the risk of cervical cancer 20.67 times more, compared to those who have had from 2 to 4 sexual partners.
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Revista Colombiana de Obstetricia y Ginecología, ISSN 0034-7434 versión impresa.
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Wang SS, Wheeler CM, Hildesheim A, Schiffman M, Herrero R, Bratti MC, et al. 2001. Human leukocyte antigen class I and II alleles and risk of cervical neoplasia: results from a population-based study in Costa Rica. J Infect Dis 184:1310-1314.
Sánchez V, Izquierdo A, Beltrán M, Viladiu P. 1997. Epidemiología del cáncer invasor de cerviz en el área sanitaria de Girona durante el período 1980-1989. Registro poblacional de cáncer de Gerona. Rev Esp Salud Pública 71:19-26.
Hart KW, Williams OM, Thelwell N, Fiander AN, Brown T, Borysiewicz LK, et al. 2001. Novel method for detection, typing and quantification of human papillomaviruses in clinical samples. J Clin Microbiol 39:3204-3212.
Ellerbrock TV, Chiasson MA, Bush TJ, Sun XW, Sawo D, Brudney K, et al. 2000. Incidence of cervical squamous intraepithelial lesions in HIV-infected women. JAMA 283:1031-1037.
Szklo M, Nieto J. 2003. Epidemiología Intermedia. Ediciones Diaz Santos, p. 26.
French PW, Coppleson M, Reid BL. 1987. Effects of sperm protamine on human cervical epithelial cells and BHK 21 cells in vitro. JR Soc Med 80:434-437.
Varghese C, Amma NS, Chitrathara K, Dhakad N, Rani P, Malathy L, et al. 1999. Risk factors for cervical dysplasia in Kerala, India. Bull World Health Organ 77: 281-3.
Molano M, Posso HJ, Weiderpass E, van den Brule AJ, Ronderos M, Franceshi S, et al. 2002. Prevalence and determinants of HPV infection among Colombian women with normal citology. Br J Cancer 87:324-333.
Munoz N, Kato I, Bosch FX, Eluf-Neto J, de San Jose S, Ascunce N, et al. 1996. Risk factor for HPV detection in middle-age women. Sex Transm Dis 23:504-510.
Félix J. Sansó Soberats; Patricia Alonso Galbán; Rosa María Torres Vida. 2010. Mortalidad por cáncer en Cuba.
Bernard W, Stewart and Alan S. Coates. 2005. Cancer prevention: a global perspective. J Clin Oncol 23:392-403.